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Marijuana And Pregnancy Study (Video) - Dr. Dreher, Phd


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ok guys. lets chill.

 

it's all good. it is a heated topic. if anybody knows, I DO. read my story again. i dont need a medical study to know my case was true.

 

 

 

 

with that being said, i haven't peeked in this topic for a couple days and it's gotten outta hand <_<

 

 

 

lmao.... i guess i gotta "moderate".

 

 

 

 

 

 

 

I just wish this test was done by more credible source.

 

 

Yes. Done be a credible health source. Not some lady in Jamaica.

 

 

yes, we understand that you doubt the credibility of the original post. we all have our own opinions as to what is "credible". if the united states government posted findings on their personal studies on MMJ/pregnancy would you believe them? are they credible to you? because to some that would be good enough...... but others would trust tyrone biggums before they trusted the white house.

 

 

 

 

Pediatrics, February 1994, Volume 93, Number 2, pp. 254-260.

American Academy of Pediatrics

 

 

Prenatal Marijuana Exposure and Neonatal Outcomes in Jamaica:

An Ethnographic Study

Melanie C. Dreher, PhD; Kevin Nugent, PhD; and Rebekah Hudgins, MA

 

 

 

From the Schools of Nursing, Education, and Public Health, the University of Massachusetts, Amherst.

Received for publication Sep 21, 1992; accepted Jun 30, 1993.

Reprint requests to (M.D.) School of Nursing, the University of Massachusetts,

111 Arnold House, Amherst, MA 01003.

Pediatrics (ISSN 0031 4005). Copyright © 1994 by the American Academy of Pediatrics.

Not credible? Some lady? You're not living up to your name, my man. <_<

 

it appears that the study was done by doctors from the university of mass. and the studies were done on patients in jamaica. i see what you were trying to say here mezz.

 

 

thanks for sharing bubblegrower. just curious, what are your opinions/feelings in regards to your teenager now smoking pot herself?

 

oh gosh.......... i could go on for hours about this. i just pray that they come to me first.

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http://www.babycenter.com/404_is-it-safe-to-smoke-marijuana-during-pregnancy_2490.bc

 

 

 

 

http://www.reuters.com/article/idUSTRE60L55L20100122

 

 

 

 

Its just as easy to find links/sources that are alot more recent that 1994 and say the exact opposite thing. I dont know either way, its just not something i think should be promoted in any means.

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The "facts" in this article are actually false, and not backed by a single scientific human study that I have ever seen.

 

"This study is bogus, read the real facts here http://press.thelancet.com/cannabis.pdf

 

Infants do return to normal after they are born and they show no different from a non cannabis smoking mother."

Its just as easy to find links/sources that are alot more recent that 1994 and say the exact opposite thing. I dont know either way, its just not something i think should be promoted in any means.

Cannabis is not for everyone, and shouldn't be used during pregnancy unless there is a need to treat a medical condition that would otherwise require the use of chemical drugs that could harm the developing baby.

 

Promoting it and considering it as a safer natural option to potentially more dangerous pharmaceutical substances for relief of things like chronic pain, morning sickness, chronic nausea, or conditions like MS for people who are pregnant are two very different things.

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Author: Mark BROWNE

Date: 12/10/2005

Source: Saanich News (.com) (British Columbia - Canada)

Copyright: © 2005 Saanich News

 

Study favours pot smoking in pregnancy

By Mark BROWNE

 

Victoria News

Oct 12 2005

 

Some people might be shocked at the idea of pregnant women smoking marijuana to deal with the nausea that comes with pregnancy.

 

But a UK-based medical publication, Journal of Complementary Therapies in Clinical Practice, has taken the idea seriously and published a study conducted by the Vancouver Island Compassion Society on the topic.

 

The Victoria-based society, which provides medicinal marijuana to people suffering from various illnesses, recently completed the study that examines the therapeutic potential of medicinal cannabis for nausea and vomiting associated with pregnancy. The study argues that marijuana is an effective method to deal with nausea and vomiting with pregnant women.

 

"It's an area that without a doubt is going to be a bit more controversial as an area of research," said Phillipe Lucas, director of the Vancouver Island Compassion Society.

 

Provincial Health Officer Dr. Perry Kendall said that marijuana likely is an effective treatment for dealing with nausea and vomiting with pregnant women.

 

"But I don't think I'd recommend smoking marijuana in pregnancy," he said.

 

There are concerns among some in the medical profession that marijuana could cause difficulty in terms of the development of the baby's brain, Kendall said. As well, it's possible that a low birth weight could result from a pregnant woman smoking the substances found in marijuana leaves, he said.

 

"So I wouldn't recommend smoking anything during pregnancy," Kendall said. Lucas conducted the research with B.C. Compassion Club Society researcher Rielle Capler, University of B.C. professor Patricia A. Janssen and University of Victoria sociologist Rachel Westfall.

 

The study was prompted by a request from Westfall who approached the Vancouver Island Compassion Society to find out how she could gain access to cannabis to conduct a study on how it might address nausea with pregnant women, Lucas said.

 

"I knew right away that, that was simply going to be an impossibility," he said.

 

There's no way, Lucas said, that the federal government would allow a clinical trial on determining if marijuana could effectively treat nausea and vomiting with pregnant women.

 

That said, Lucas decided to move forward with a survey/study to determine if women who smoked marijuana while they were pregnant found that it dealt with the nausea and vomiting.

 

The survey shows that 92 per cent of respondents considered marijuana to be either "extremely effective" or "effective" as a therapy for nausea and vomiting (or morning sickness).

 

The study also focused on a particularly severe form of nausea and vomiting associated with pregnancy called hyperenesisgragidarum.

 

"It's characterized as extremely severe nausea that affects two per cent of women who go through pregnancy," Lucas said.

 

As it currently stands, there aren't any effective pharmaceutical treatments available to treat that condition, he said.

 

The study also focused on how effective marijuana is in treating nausea in general. The vast majority of respondents indicated that marijuana is an effective therapy for nausea (93 per cent), vomiting (75 per cent) and as an appetite stimulant (95 per cent).

 

At the same time, the suggestion that marijuana is an effective treatment for nausea is apparent from talking to clients at the Vancouver Island Compassion Society, Lucas said.

 

"It's something that we see - everyday."

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Women who smoke marijuana during pregnancy may impair their baby's growth and development in the womb, a new study suggests.

 

HEALTH

 

Poor fetal growth and reduced head circumference at birth are linked to an increased risk of problems with thinking, memory and behavior in childhood. Cigarette smoking during pregnancy is known to impair fetal growth, but studies on the potential effects of marijuana have been inconclusive.

 

For the new study, researchers in the Netherlands followed more than 7,000 pregnant women, 3 percent of whom acknowledged smoking marijuana at least during early pregnancy. They found that babies born to marijuana users tended to weigh less and have smaller heads than other infants.

 

What's more, the study found, the longer a woman had used marijuana during pregnancy, the stronger the impact on birth size -- suggesting that the drug itself was to blame.

 

And while most marijuana users in the study also smoked cigarettes, the drug appeared to have effects over and above those of tobacco. In fact, marijuana showed stronger effects on birth size than tobacco, the investigators report in the Journal of the American Academy of Child and Adolescent Psychiatry.

 

The findings suggest that marijuana use, even restricted to early pregnancy, may have irreversible effects on fetal growth, write the researchers, led by Hannan El Marroun of Erasmus University Medical Center in Rotterdam.

 

To prevent this, they add, women who smoke marijuana should quit before becoming pregnant.

 

The study included almost 7,500 pregnant women who were surveyed on their use of alcohol, tobacco and drugs, and had ultrasounds to chart fetal growth during the first, second and third trimesters.

 

Overall, 214 women said they had used marijuana before and during early pregnancy; 81 percent quit after learning they were pregnant, but 41 women continued to smoke marijuana throughout pregnancy.

 

The researchers found that, on average, marijuana users gave birth to smaller babies, particularly those who had used throughout pregnancy.

 

Women who had smoked only during early pregnancy had babies who were

 

156 grams -- about 5.5 ounces -- lighter than infants born to women who had not used the drug. Women who had continued to smoke past early pregnancy had babies who were 277 grams, or nearly 10 ounces, smaller.

 

Based on ultrasound, marijuana use only in early pregnancy impaired fetal growth by about 11 grams per week, while use throughout pregnancy slowed fetal growth by roughly 14 grams per week. That compared with a deficit of 4 grams per week with tobacco use, the researchers found.

 

Similar patterns were seen when the researchers looked at fetal head circumference.

 

According to El Marroun's team, mothers' marijuana use could stunt fetal growth for several reasons. Like tobacco smoking, it may deprive the fetus of oxygen. It is also possible that the byproducts of marijuana directly affect the developing nervous and hormonal systems of the fetus.

 

Finally, the researchers note, pregnant women who use marijuana may have other factors in their lives - such as a less-than-healthy diet or chronic stress -- that could contribute to poor fetal growth.

 

SOURCE: Journal of the American Academy of Child and Adolescent Psychiatry, December 2009.

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Women who smoke marijuana during pregnancy may impair their baby's growth and development in the womb, a new study suggests.

 

HEALTH

 

Poor fetal growth and reduced head circumference at birth are linked to an increased risk of problems with thinking, memory and behavior in childhood. Cigarette smoking during pregnancy is known to impair fetal growth, but studies on the potential effects of marijuana have been inconclusive.

 

For the new study, researchers in the Netherlands followed more than 7,000 pregnant women, 3 percent of whom acknowledged smoking marijuana at least during early pregnancy. They found that babies born to marijuana users tended to weigh less and have smaller heads than other infants.

 

What's more, the study found, the longer a woman had used marijuana during pregnancy, the stronger the impact on birth size -- suggesting that the drug itself was to blame.

 

And while most marijuana users in the study also smoked cigarettes, the drug appeared to have effects over and above those of tobacco. In fact, marijuana showed stronger effects on birth size than tobacco, the investigators report in the Journal of the American Academy of Child and Adolescent Psychiatry.

 

The findings suggest that marijuana use, even restricted to early pregnancy, may have irreversible effects on fetal growth, write the researchers, led by Hannan El Marroun of Erasmus University Medical Center in Rotterdam.

 

To prevent this, they add, women who smoke marijuana should quit before becoming pregnant.

 

The study included almost 7,500 pregnant women who were surveyed on their use of alcohol, tobacco and drugs, and had ultrasounds to chart fetal growth during the first, second and third trimesters.

 

Overall, 214 women said they had used marijuana before and during early pregnancy; 81 percent quit after learning they were pregnant, but 41 women continued to smoke marijuana throughout pregnancy.

 

The researchers found that, on average, marijuana users gave birth to smaller babies, particularly those who had used throughout pregnancy.

 

Women who had smoked only during early pregnancy had babies who were

 

156 grams -- about 5.5 ounces -- lighter than infants born to women who had not used the drug. Women who had continued to smoke past early pregnancy had babies who were 277 grams, or nearly 10 ounces, smaller.

 

Based on ultrasound, marijuana use only in early pregnancy impaired fetal growth by about 11 grams per week, while use throughout pregnancy slowed fetal growth by roughly 14 grams per week. That compared with a deficit of 4 grams per week with tobacco use, the researchers found.

 

Similar patterns were seen when the researchers looked at fetal head circumference.

 

According to El Marroun's team, mothers' marijuana use could stunt fetal growth for several reasons. Like tobacco smoking, it may deprive the fetus of oxygen. It is also possible that the byproducts of marijuana directly affect the developing nervous and hormonal systems of the fetus.

 

Finally, the researchers note, pregnant women who use marijuana may have other factors in their lives - such as a less-than-healthy diet or chronic stress -- that could contribute to poor fetal growth.

 

SOURCE: Journal of the American Academy of Child and Adolescent Psychiatry, December 2009.

 

Finally, the researchers note, pregnant women who use marijuana may have other factors in their lives - such as a less-than-healthy diet or chronic stress -- that could contribute to poor fetal growth.

 

There are so many variables that could skew an unscientific study like this one way or another, not the least of all women lying about not smoking and all the other millions of variables that come into play when it comes to determining birth weight and fetal development. Just because someone decides that a study might suggest something doesn't mean you should look at it as the truth, because it's far from a scientific finding.

 

Keep in mind that even when we think science proves something it's not uncommon for us to learn something new years later that disproves everything that we once thought we knew about that subject. We should never stop being willing to consider all possibilities and look at things objectively from every angle.

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This sums up the debate pretty well:

 

http://www.mothering.com/pregnancy-birth/use-of-marijuana-during-pregnancy

 

Use of Marijuana During Pregnancy

 

Warnings that marijuana causes birth defects date back to the late 1960s.1 Some researchers claimed to have found chromosomal abnormalities in blood cells taken from marijuana users. They predicted that young men and women who used marijuana would produce deformed babies.2 Although later studies disproved this theory,3 some current drug education materials still claim that genetic damage is passed on by marijuana users to their children.4

 

Today, researchers look for a direct effect of THC [for tetrahydrocannabinol, either of two physiologically active isomers, C21H30O2, from hemp plant resin] on the fetus. In animal studies, THC has been shown to produce spontaneous abortion, low birth weight, and physical deformities—but only with extremely large doses, only in some species of rodents, and only when THC is given at specific times during pregnancy.5 Because the effects of drugs on fetal development differ substantially across species,6 these studies have little or no relevance to humans. Studies with primates show little evidence of fetal harm from THC.7 In one study, researchers exposed chimpanzees to high doses of THC for up to 152 days and found no change in the sexual behavior, fertility, or health of their offspring.8

 

Dozens of studies have compared the newborn babies of women who used marijuana during pregnancy with the babies of women who did not. Mainly, they have looked for differences in birth weight, birth length, head circumference, chest circumference, gestational age, neurological development, and physical abnormalities. Most of these studies, including the largest study to date with a sample of over twelve thousand women,9 have found no differences between babies exposed to marijuana prenatally and babies not exposed.10 Given the large number of studies and the large number of measures, some differences are likely to occur by chance. Indeed, researchers have found differences in both directions. In some studies, the babies of marijuana users appear healthier and hardier.11 In others, researchers have found more adverse outcomes in the babies of marijuana users.12

 

When adverse outcomes are found, they are inconsistent from one study to another, always relatively minor, and appear to have no impact on infant health or mortality.13 For example, in one recent study, researchers reported a statistically significant effect of marijuana on birth length. The marijuana-exposed babies, on average, were less than two-tenths of one inch shorter than babies not exposed to marijuana.14 Another study found a negative effect of marijuana on birth weight, but only for White women in the sample.15 In a third study, marijuana exposure had no effect on birth weight, but a small negative effect on gestational age.16 Overall, this research indicates no adverse effect of prenatal marijuana exposure on the physical health of newborns.

 

Researchers have also examined older children for the effects of prenatal exposure to marijuana. A study of one-year-olds found no differences between marijuana-exposed and nonexposed babies on measures of health, temperament, personality, sleeping patterns, eating habits, psychomotor ability, physical development, or mental functioning.17 In two studies, one of three-year-olds,18 the other of four-year-olds,19 there was no effect of prenatal marijuana exposure on children’s overall IQ test scores. However, in the first study, when researches looked at Black and White children separately, they found, among Black children only, slightly lower scores on two subscales of the IQ test. On one subscale, it was children exposed to marijuana only during the first trimester who scored lower. On the other subscale, it was children exposed during the second trimester who scored lower.20 In neither case did the frequency or quantity of mothers’ marijuana use affect the outcomes. This makes it highly unlikely they were actually caused by marijuana. Nonetheless, this study is now cited as evidence that using marijuana during pregnancy impairs the intellectual capacity of children.21

 

Also widely cited are two recent case-control studies describing a relationship between marijuana use by pregnant women and two rare forms of cancer in their children. A case-control study compares people with a specific disease (the case sample) to people without the disease (the control sample). Using this method, researchers identify group differences in background, environment, lifestyle, drug use, diet, and the like that are possible causes of the disease.

 

A study of children with non-lymphoblastic leukemia reported a tenfold greater risk related to their mothers’ use of marijuana during pregnancy.22 A second study reported a threefold greater risk of rhabdomyosarcoma.23 These calculations were based on women’s reports that they used marijuana at some point during pregnancy. In the first study, ten out of the 204 case-group mothers (5 percent) reported marijuana use, compared to one out of the 204 control-group mothers (0.5 percent). In the second study, 8 percent of case-group mothers reported using marijuana, compared to 4.3 percent of controls.

 

These studies do not prove that marijuana use by pregnant women causes cancer in their children. They report a statistical association based solely on women’s self-reports of marijuana use. It is likely that both groups of mothers underreported marijuana use; in other studies, researchers have found that marijuana use by pregnant women typically ranges from 10 to 30 percent.24 There is reason to suspect greater underreporting by control-group mothers, who were randomly selected and questioned about their marijuana use on the telephone. Because the mothers of the sick children were trying to help researches identify the cause of their children’s disease, they had more reason to be honest about their illegal drug use.

 

Like all case-control studies, these two studies identified many differences between case-group mothers and control-group mothers, all of which could possibly lead scientists to discover the cause of these rare forms of cancer. Other factors associated with childhood rhabdomyosarcoma include low socioeconomic status, fathers’ cigarette smoking, a family history of allergies, children’s exposure to environmental chemicals, childhood diets that include organ meats, mothers’ use of antibiotics during pregnancy, mothers being over age thirty at the time of the child’s birth, overdue delivery, and the child having had fewer immunizations.25 Without additional research, none of the factors that are statistically associated with childhood cancer can be identified as causes of childhood cancer. At this time, there is no corroborative evidence to link marijuana with cancer. In fact, in a recent study, researchers found significantly lower rates of cancer in rats and mice following two years of exposure to extremely large doses of THC.26

 

Since 1978, psychologist Peter Fried and his colleagues have collected longitudinal data on prenatal marijuana exposure as part of the Ottawa Prenatal Prospective Study (OPPS). Over the years, these researchers have administered hundreds of tests to the same group of children, assessing their physical development, psychomotor ability, emotional and psychological adjustment, cognitive functioning, intellectual capacity, and behavior.

 

Out of all the OPPS studies and all the tests given, researchers have found very few differences between marijuana-exposed and nonexposed children. At age one, researchers found that marijuana-exposed infants scored higher on one set of cognitive tests.27 At age three, the children of moderate marijuana users (one to five joints per week during pregnancy) had higher scores on one test of psychomotor ability.28 At age four, the children of women who smoked marijuana heavily during pregnancy (an average of nineteen joints per week) scored lower on one subscale of one cognitive test.29 However, at ages five and six, this difference was no longer present.30 When the children were six, the researchers added several new measures of “attentional behavior.” The children of heavy marijuana users scored lower on one computer-based test of “vigilance.”31 Eleven new psychological and cognitive tests, administered to six- to nine-year-olds, showed no statistically significant differences between the children of marijuana users and nonusers. Parents rated marijuana-exposed children as having more “conduct problems,” but this difference disappeared after the researchers controlled for confounding variables.32

 

Despite the overwhelming similarities in the children of marijuana users and nonusers, in their published reports OPPS researchers consistently highlight the occasional negative finding. Fried believes that these findings underestimate the harms of prenatal marijuana exposure. He suggests that “more sensitive measures” are needed because:

 

instruments that provide a general description of cognitive abilities may not be capable of identifying nuances in neuro-behaviour that may discriminate between the marijuana-exposed and non-marijuana exposed children. . . . Tests that examine specific characteristics that may underline cognitive performance may be more appropriate and successful.33

 

Recently, Fried predicted that a new test of “executive function” would reveal marijuana-related deficits in preteen youngsters.34 A short time later, Fried announced that preliminary analysis of his data showed this effect was present.35 Almost immediately, his announcement appeared in U.S. government reports as evidence of marijuana’s harm to the fetus.36 Additional reports of harm based on the OPPS sample, which now includes fewer than thirty marijuana-exposed children, may be forthcoming—despite the fact that, according to Fried, the consequences of prenatal drug exposure typically diminish as children get older.37

After controlling for known confounding variables, Fried estimates that prenatal drug exposure accounts for 8 percent or less of the variance in children’s scores on developmental and cognitive tests—and this estimate is for alcohol, tobacco, and marijuana combined.38 In essentially all studies, marijuana contributes less than alcohol or tobacco.39 In addition, the findings differ from one study to another, and show no consistent relationship of fetal harm to either the timing or degree of marijuana exposure. While it is sensible to advise women to abstain from all drugs during pregnancy, the weight of current scientific evidence suggests that marijuana does not directly harm the human fetus.

 

NOTES

1. F. Hecht et al., “Lysergic-Acid-Diethylamide and Cannabis as Possible Teratogens in Man,” Lancet 2 (1968): 1087. G. Carakushansky et al., “Lysergide and Cannabis as Possible Teratogens in Man,” Lancet 1 (1969): 150–151.

 

2. T. H. Maugh, “Marihuana: The Grass May No Longer Be Greener,” Science 185 (1974): 683–685.

 

3. S. Matsuyama and L. Jarvik, “Effects of Marihuana on the Genetic and Immune Systems,” in R. C. Petersen (ed.), Marihuana Research Findings, 1976 (Rockville, MD: National Institute on Drug Abuse, 1977), 179–193. K. Morishima, “Effects of Cannabis and Natural Cannabinoids on Chromosomes and Ova,” in M. C. Braude and J. L. Ludford (eds.), Marijuana Effects on the Endocrine and Reproductive Systems (Rockville, MD: National Institute on Drug Abuse, 1984), 25–45.

 

4. Parents Resource Institute for Drug Education, Marijuana: Effects on the Male, (Atlanta, GA: PRIDE, 1996). W. R. Spence, Marijuana: Its Effects and Hazards (Waco, TX: Health Edco, undated). Peggy Mann, The Sad Story of Mary Wanna (New York: Woodmere Press, 1988), 30.

 

5. J. Herclerode, “The Effect of Marijuana on Reproduction and Development,” in R. C. Petersen (ed.), Marijuana Research Findings: 1980 (Rockville, MD: National Institute on Drug Abuse, 1980), 137–166. E. L. Abel, “Effects of Prenatal Exposure to Cannabinoids,” in T. M. Pinkert (ed.), Current Research on the Consequences of Maternal Drug Abuse (Rockville, MD: National Institute on Drug Abuse, 1985), 20–35. D. Hutchings and D. Dow-Edwards, “Animal Models of Opiate, Cocaine, and Cannabis Use,” Clinics in Perinatology 18 (1991): 1–22. M. Behnke and F. D. Eyler “The Consequences of Prenatal Substance Use for the Developing Fetus, Newborn, and Young Child,” International Journal of the Addictions 28 (1993): 1341–1391. T. Wenger et al., “Effects of Delta-9-Tetrahydrocannabinol on Pregnancy, Puberty, and the Neuroendocrine System,” in L. Murphy and A. Bartke (eds.), Marijuana/Cannabinoids: Neurobiology and Neurophysiology (Boca Raton, FL: CRC Press, 1992), 539–560.

 

6. A. M. Rudolph, “Animal Models for Study of Fetal Drug Exposure,” in C. N. Chiang and C. C. Lee (eds.), Prenatal Drug Exposure: Kinetics and Dynamics (Rockville, MD: National Institute on Drug Abuse, 1985), 5–16

 

7. P. A. Fried, “Postnatal Consequences of Maternal Marijuana Use,” in T. M. Pinkert (ed.), Current Research on the Consequences of Maternal Drug Abuse (Rockville, MD: National Institute on Drug Abuse, 1985), 61–72. M. S. Golub et al., “Peer and Maternal Social Interaction Patterns in Offspring of Rhesus Monkeys Treated Chronically with Delta-9-Tetrahydrocannabinol,” in S. Agurell, The Cannabinoids: Chemical, Pharmacological, and Therapeutic Aspects (Orlando, FL: Academic Press, 1984), 657–667. J. Herclerode (1980), see Note 5.

 

8. D. M. Grilly et al., “Observations on the Reproductive Activity of Chimpanzees Following Long-Term Exposure to Marijuana,” Pharmacology 11 (1974): 304–307.

 

9. S. Linn et al., “The Association of Marijuana use with Outcome of Pregnancy,” American Journal of Public Health 73 (1983): 1161–1164.

 

10. P. H. Shiono et al., “The Impact of Cocaine and Marijuana Use on Low Birth Weight and Preterm Birth: A Multicenter Study,” American Journal of Obstetrics and Gynecology 172 (1995): 19–27. E. M. Knight et al., “Relationships of Serum Illicit Drug Concentrations During Pregnancy to Maternal Nutritional Status,” Journal of Nutrition 124 (1994): 973–980S. K. Tennes and C. Blackard, “Maternal Alcohol Consumption, Birthweight, and Minor Physical Abnormalities,” American Journal of Obstetrics and Gynecology 138 (1980): 774–780. J. Hayes et al., “Newborn Outcomes with Maternal Marijuana Use in Jamaican Women,” Pediatric Nursing 14 (1988): 107–110. P. A. Fried and C. M. O’Connell, “A Comparison of the Effects of Prenatal Exposure to Tobacco, Alcohol, Cannabis and Caffeine on Birth Size and Subsequent Growth,” Neurotoxicology and Teratology 9 (1987): 79–85. C. M. O’Connell and P. A. Fried, “An Investigation of Prenatal Cannabis Exposure and Minor Physical Anomalies in a Low Risk Population,” Neurobehavioral Toxicology and Teratology 6 (1984): 345–350. G. A. Richardson et al., “The Effect of Prenatal Alcohol, Marijuana and Tobacco Exposure on Neonatal Behavior,” Infant Behavioral Development 12 (1989): 199–209. S. Astley, “Analysis of Facial Shape in Children Gestationally Exposed to Marijuana, Alcohol, and/or Cocaine,” Pediatrics 89 (1992): 67–77. F. R. Witter and J. R. Niebyl, “Marijuana Use in Pregnancy and Pregnancy Outcome,” American Journal of Perinatology 7 (1990): 36–38.

 

11. M. C. Dreher et al., “Prenatal Exposure and Neonatal Outcomes in Jamaica: An Ethnographic Study,” Pediatrics 93 (1994): 254–60. K. Tennes et al., “Marijuana: Prenatal and Postnatal Exposure in the Human,” in T. M. Pinkert (ed.), Current Research on the Consequences of Maternal Drug Abuse (Rockville, MD: National Institute on Drug Abuse, 1985), 48–60.

 

12. E. E. Hatch and M. B. Bracken, “Effect of Marijuana Use in Pregnancy on Fetal Growth,” American Journal of Epidemiology 124 (1986): 986–993. J. Kline et al., “Cigarettes, Alcohol and Marijuana: Varying Associations with Birthweight,” International Journal of Epidemiology 16 (1987): 44–51. B. Zuckerman et al., “Effects of Maternal Marijuana and Cocaine Use on Fetal Growth,” New England Journal of Medicine 320 (1989): 762–768. P. A. Fried et al., “Marijuana Use During Pregnancy and Decreased Length of Gestation,” American Journal of Obstetrics and Gynecology 150 (1984): 23–26. R. Hingson et al., “Effects of Maternal Drinking and Marijuana Use on Fetal Growth and Development,” Pediatrics 70 (1982): 539–546. P. A. Fried and J. E. Makin, “Neonatal Behavioral Correlates of Prenatal Exposure to Marijuana, Cigarettes and Alcohol in a Low Risk Population,” Neurotoxicology and Teratology 9 (1987): 1–7. M. D. Cornelius et al., “Prenatal Tobacco and Marijuana Use Among Adolescents: Effects on Offspring Gestational Age, Growth, and Morphology,” Pediatrics 95 (1995): 738–743. N. Day et al., “Prenatal Marijuana Use and Neonatal Outcome,” Neurotoxicology and Teratology13 (1991): 329–334.

 

13. N. L. Day and G. A. Richardson, “Prenatal Marijuana Use: Epidemiology, Methodologic Issues, and Infant Outcome,” Clinics in Perinatology 18 (1991): 77–91. G. A. Richardson et al., “The Impact of Marijuana and Cocaine Use on the Infant and Child,” Clinical Obstetrics and Gynecology 36 (1993): 302–318. M. D. Cornelius et al. (1995), see Note 12. C. D. Coles et al., “Effects of Cocaine, Alcohol, and Other Drug Use in Pregnancy on Neonatal Growth and Neurobehavioral Status,” Neurotoxicology and Teratology 14 (1992): 22–33.

 

14. N. Day et al. (1991), see Note 12.

 

15. E. E. Hatch and M. B. Bracken (1986), see Note 12.

 

16. P. A. Fried et al. (1984), see Note 12.

 

17. K. Tennes et al. (1985), see Note 11.

 

18. N. L. Day et al., “Effect of Prenatal Marijuana Exposure on the Cognitive Development of Offspring at Age Three,” Neurotoxicology and Teratology 16 (1994): 169–175.

 

19. A. P. Streissguth, et al., “IQ at Age 4 in Relation to Maternal Alcohol Use and Smoking During Pregnancy,” Developmental Psychology 25 (1989): 3–11.

 

20. See Note 18.

 

21. Center on Addiction and Substance Abuse, Legalization: Panacea or Pandora’s Box (New York, 1995). Drug Watch Oregon, Marijuana Research Review 2 (1995): 4.

 

22. L. L. Robison et al., “Maternal Drug Use and Risk of Non-Lymphoblastic Leukemia Among Offspring,” Cancer 63 (1989): 1904–1911.

 

23. S. Grufferman et al., “Parents’ Use of Cocaine and Marijuana and Increased Risk of Rhabdomyosarcoma in Their Children,” Cancer Causes and Control 4 (1993): 217–224.

 

24. N. L. Day et al., “The Epidemiology of Alcohol, Marijuana and Cocaine Use Among Women of Childbearing Age and Pregnant Women,” Clinical Obstetrics and Gynecology 36 (1993): 232–245.

 

25. S. Grufferman et al., “Environmental Factors in the Etiology of Rhabdomyosarcoma in Childhood,” Journal of the National Cancer Institute 68 (1982): 107–113.

 

26. National Toxicology Program, Toxicology and Carcinogenesis: Studies of 1-Trans-Delta-9-Tetrahydrocannabinol in F344/N Rats and B6c3F1 Mice (Rockville, MD: U.S. Department of Health and Human Services, 1996).

 

27. P. A. Fried and B. Watkinson, “12- and 24-Month Neurobehavioral Follow-Up of Children Prenatally Exposed to Marijuana, Cigarettes and Alcohol,” Neurotoxicology and Teratology 10 (1988): 305–313.

 

28. P. A. Fried and B. Watkinson, “36- and 48-Month Neurobehavioral Follow-Up of Children Prenatally Exposed to Marijuana, Cigarettes and Alcohol,” Developmental and Behavioral Pediatrics 11 (1990): 49–58.

 

29. Ibid.

 

30. P. A. Fried et al., “60- and 72-Month Follow-Up of Children Prenatally Exposed to Marijuana, Cigarettes, and Alcohol: Cognitive and Language Assessment,” Journal of Developmental and Behavioral Pediatrics 13 (1992): 383–391.

 

31. P. A. Fried et al., “A Follow-Up Study of Attentional Behavior in 6-Year-Old Children Exposed Prenatally to Marijuana, Cigarettes, and Alcohol,” Neurotoxicology and Teratology 14 (1992): 299–311.

 

32. C. M. O’Connell and P. A. Fried, “Prenatal Exposure to Cannabis: A Preliminary Report of Postnatal Consequences in School-Age Children,” Neurotoxicology and Teratology 13 (1991): 631–639.

 

33. P. A. Fried, “Prenatal Exposure to Marijuana and Tobacco During Infancy, Early and Middle Childhood: Effects and Attempts at a Synthesis,” Archives of Toxicology 17 (1995): 240–241.

 

34. P. A. Fried, “The Ottawa Prenatal Prospective Study (OPPS): Methodological Issues and Findings—It’s Easy to Throw the Baby Out With the Bath Water,” Life Sciences 56 (1995): 2159–2168.

 

35. National Conference on Marijuana Use: Prevention, Treatment, and Research, sponsored by the National Institute on Drug Abuse (Arlington, VA: July 1995).

 

36. Center for Substance Abuse Prevention, “Marijuana: Its Uses and Effects,” Prevention Pipeline 8, no. 5 (1995): 3–5.

 

37. P. A. Fried, “Prenatal Exposure to Tobacco and Marijuana: Effects During Pregnancy, Infancy, and Early Childhood,” Clinical Obstetrics and Gynecology 36 (1993): 319–337.

 

38. Ibid.

 

39. P. A. Fried, “Cigarettes and Marijuana: Are There Measurable Long-Term Neurobehavioral Teratogenic Effects?” Neurotoxicology 10 (1989): 577–584. N. Day et al., “The Effects of Prenatal Tobacco and Marijuana Use on Offspring Growth from Birth through 3 Years of Age,” Neurotoxicology and Teratology 14 (1992): 407–414. H. M. Barr et al., “Infant Size at 8 Months of Age: Relationship to Maternal Use of Alcohol, Nicotine, and Caffeine During Pregnancy,” Pediatrics 74 (1984): 336–341. P. A. Fried and B. Watkinson (1990), see Note 28. A. P. Streissguth et al. (1989), see Note 19. M. D. Cornelius et al. (1995), see Note 12. J. Kline et al. (1987), see Note 12. P. A. Fried (1995), see Note 33.

 

This article is excerpted from Marijuana Myths, Marijuana Facts: A Review of the Scientific Evidence by Lynn Zimmer, PhD, and John P. Morgan, MD (New York: Drug Policy Alliance, 1997). Permission to reprint this article was granted by the Drug Policy Alliance, the nation’s leading organization working to end the war on drugs. For more information, visit www.drugpolicy.org.

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